by Fed Up Texas Chick, The Tenpenny Report:
“Approving a vaccine, utilizing novel RNA technology without extensive testing is extremely dangerous. The vaccine could be a bioweapon and even more dangerous than the original infection.”
Introduction
Dr. Tenpenny was one of the first people to say this in 2021 when she wrote 40 Mechanisms of Injury. Long ago, she said that injecting synthetically made SARS-CoV-2 spike protein into the entire population greatly increased the risk of long-term side effects, and that risks of developing an autoimmune illness will remain for an unknown period of time. With B-cell priming and irreversible genetic manipulation, the risk for developing chronic illness is great. The spike protein can bind to the surface of the vaccine recipient’s cells. This spike protein then becomes a potential receptor for other more aggressive or more dangerous infectious agents. SARS-CoV-2 is the only coronavirus with a prion-like domain found in the receptor-binding domain of the S1 region of the spike protein. SARS-CoV-2 demonstrates a 10- to 20-fold higher affinity for ACE2 receptor, their primary binding site, than SARS-CoV and other common coronaviruses.
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Dr. Tenpenny was right and sadly we are seeing unprecedented levels of chronic and autoimmune diseases. As prion disease become more prevalent. Here is your opportunity to increase your knowledge and understanding of prions and their potential threat to your help. Investigative Journalist Maryam Henein recently published this piece on her prion research and we are reprinting hit here for you.
Maryam’s Article
For the past several months, I have been researching prions in the cervid population. I am still in the information-gathering phase, but I first touched upon misfolded proteins in my eBook, UNDERSTANDING THE MECHANISMS OF THE COVID-19 “VACCINES.”
Is the spike protein a self-replicating amyloid? We will get into it in tomorrow’s show.
Dr. Kevin McCairn is a 25-year veteran of clinical neuroscience, specializing in movement and neuropsychiatric disorders in non-human primates. He was the Principal Investigator at the Korea Brain Research Institute. His notable contributions to the field include the discovery of brain network pathologies in neurodegenerative conditions, e.g., Parkinson’s Disease, and how deep brain stimulation (the gold-standard surgical treatment) works to treat Parkinsonian symptoms.
He is credited for the development of the world’s first non-human primate model of Tourette syndrome, leading to next-generation neurosurgical treatments for both Tourette’s and neuropsychiatric disorders such as obsessive-compulsive disorder. In early 2020, Dr. McCairn was the first to publicly raise the specter of neurological and potential prion disorders related to SARS-CoV-2.
Prions And The Rona JibJabs
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Prions, the enigmatic proteins at the heart of neurodegenerative diseases, trace their origins back to a peculiar discovery made in the 18th century. The story begins with scrapie, a mysterious ailment that plagued sheep in Europe for centuries. Farmers observed afflicted sheep exhibiting peculiar behaviors, such as intense itching, nervousness, and a tendency to scrape against fences or rocks, hence the name “scrapie.”
What was the origin of scrapies, I wondered. Does it differ from what the literature says? Given my experience studying the origins of diseases, who knows? I will be taking a deeper look in an upcoming Substacl on deer zombie disease and prions.
Probably all diseases are the result of some imbalance, whether natural or unnatural, but it’s always possible to consider that maybe a lost civilization was engaging in fuckery like we are now, so who knows,” A.W Finnegan, the author of The Sleeper Agent, recently told me Btw, I interviewed Adam last week about the origins of Lyme and his in-depth book.
It wasn’t until the 20th century that scientists claim they began unraveling the true nature of scrapie. In the 1960s, American biologist Tikvah Alper proposed the existence of an unconventional infectious agent that could replicate without containing nucleic acid, the genetic material found in all other known pathogens. This hypothesis challenged the established dogma of molecular biology, which dictated that genes were essential for reproduction.
Stanley B. Prusiner’s work in the 1980s provided the missing pieces of the puzzle. Prusiner coined the term “prion” (short for “proteinaceous infectious particle”) to describe the mysterious agent responsible for scrapie and other similar diseases. He proposed that prions were misfolded proteins capable of inducing normal proteins to adopt the aberrant conformation, leading to a cascade of misfolding and aggregation in the brain, ultimately causing neurodegeneration.
The discovery of prions revolutionized the understanding of infectious diseases, challenging the traditional notions of pathogens and prompting a paradigm shift in molecular biology. Unlike conventional viruses or bacteria, prions lack genetic material entirely and rely solely on the structural alteration of proteins to propagate. This unique mode of transmission makes prion diseases particularly insidious, as prion disease takes years to express itself and can spread through exposure via contaminated tissues.
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