by Angelo DePalma, Ph.D., Childrens Health Defense:
A 2023 study admitted that the COVID-19 mRNA vaccines cause myocarditis, but claimed the COVID-19 virus was even more damaging than the vaccine. A recent, more detailed review of their data, however, showed the opposite is likely true.
Despite the known side effects of mRNA COVID-19 vaccines, some studies (here, here and here) and health websites (here, here and here) argue that whatever vaccination’s adverse outcomes, being unvaccinated is worse.
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In one such study, Dr. Christian Mueller and his co-authors concluded the COVID-19 virus — not the vaccine — was responsible for more myocarditis, or heart muscle damage, than the vaccine.
However, when Rainer Johannes Klement, Ph.D., a physicist at Leopoldina Hospital in Schweinfurt, Germany, and Harald Walach, a clinical psychologist and head of the Change Health Institute in Basel, Switzerland, reanalyzed Mueller’s data they found that while coronaviruses might cause myocarditis, the COVID-19 vaccines cause at least as much or more.
The Klement paper appeared in the Feb. 1 edition of The Egyptian Health Journal.
Mueller’s study
Mueller set out to quantify and compare myocarditis in vaxed versus unvaxed subjects and to explain possible mechanisms.
To explore these mechanisms, the researchers tested subjects for antibodies against interleukin-1 receptor antagonist (IL-1RA), the SARS-CoV-2-nucleoprotein, the viral spike protein and 14 inflammatory cytokines.
Since none of these measures differed between study groups, the “mechanism” issue was unresolved.
To assess myocarditis investigators tested 777 hospital workers (median age 37, 69.5% women) for cardiac troponin T one and three days after they received an mRNA-1273 booster. Cardiologists typically prescribe this test after a suspected heart attack to quantify the extent and duration of heart damage.
Of the 40 subjects (5.1%) with elevated troponin on Day 3, 22 (2.8%) were diagnosed with myocarditis, with 20 cases occurring in women and two in men.
The researchers reported that among these subjects troponin elevations were mild and temporary and did not involve abnormalities as determined by electrocardiogram. No patients experienced “major adverse cardiac events” within 30 days of receiving the shot.
- COVID-19 associates with a substantially higher risk for myocarditis that [sic] mRNA vaccination …
- Myocarditis related to COVID-19 infection has shown a higher mortality than myocarditis related to mRNA vaccination.
- Before the COVID-19 vaccine were [sic] available, the incidence and extent of myocardial injury associated with COVID-19 infection was [sic] much higher than observed in this active surveillance study after booster vaccination.
One of the Mueller co-authors had commercial ties to diagnostics companies. Another had previously been compensated by diagnostics and vaccine manufacturers. Mueller had relationships with diagnostics, pharmaceutical and vaccine companies at the time he wrote the paper.
Where did Mueller go wrong?
One way to measure treatment effects is to compare an outcome, for example, blood pressure, in the same subjects before and after the treatment and report before-and-after results.
Although this option was known to medical researchers and available to him, Mueller did not take advantage of it — either because he did not think to measure pre-booster troponin levels or chose not to report them for some reason, perhaps because they did not align with his other results.
Instead, his team took an approach that required two well-matched study groups. Although Mueller claimed placebos and controls met this requirement they differed on the feature that mattered most: heart health.
Vaccinated subjects with current or recent heart issues were excluded from the study, while all control subjects had just entered the hospital with heart symptoms and were therefore already at greater risk for myocarditis.
Klement and Walach found more anomalies in the Mueller paper.
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