by Sean Adl-Tabatabai, The Peoples Voice:
According to new data, one of the more disturbing side effects of the COVID-19 mRNA vaccines is “contagious dementia”, which scientists say is transmissible via prions.
In a 2021 interview with Stephanie Seneff, Ph.D., she explained why she believed the COVID-19 shots may eventually result in an avalanche of neurological prion-based diseases such as Alzheimer’s.
She also published a scientific paper detailing those mechanisms in the May 10, 2021, issue of the International Journal of Vaccine Theory.
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As she explained in that paper:
“A paper published by J. Bart Classen (2021) proposed that the spike protein in the mRNA vaccines could cause prion-like diseases, in part through its ability to bind to many known proteins and induce their misfolding into potential prions.
“Idrees and Kumar (2021) have proposed that the spike protein’s S1 component is prone to act as a functional amyloid and form toxic aggregates … and can ultimately lead to neurodegeneration.”
Infowars.com reports: In summary, the take-home from Seneff’s paper is that the COVID-19 shots, offered to hundreds of millions of people, are instruction sets for your body to make a toxic protein that will eventually wind up concentrated in your spleen, from where prion-like protein instructions will be sent out, leading to neurodegenerative diseases.
What are prions?
The term “prion” derives from “proteinaceous infectious particle.” Prions are known to cause a variety of neurodegenerative diseases in animals and humans, such as CJD in humans, bovine spongiform encephalopathy (BSE or “mad cow disease”) in cattle and chronic wasting disease in deer and elk.
These diseases are collectively referred to as transmissible spongiform encephalopathies. They’re characterized by long incubation periods, brain damage, the formation of holes in the brain giving it a sponge-like appearance and failure to induce an inflammatory response.
In short, prions are infectious agents composed entirely of a protein material that can fold in multiple, structurally distinct ways, at least one of which is transmissible to other prion proteins, leading to a disease that is similar to viral infections but without nucleic acids.
Unlike bacteria, viruses and fungi, which contain nucleic acids (DNA or RNA) that instruct their replication, prions propagate by transmitting their misfolded protein state to normal variants of the same protein.
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